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Cells accumulate mutations with age
Splicing alterations are abundant in blood cancer
Multiple factors limit the study of splicing fact mutations in human samples
GoT-Splice: Genotype, transcriptomes and splicing at single-cell resolution
CITE-seq and GoT improve cell-type identification
Biotin selection improves recovery of long-re transcripts
Mutant cells show increased 3' splicing
MDS patients present cell-type specific splicing
Splicing factor mutations are also abunda in clonal hematopoiesis (CH)
Conclusions Series