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Two major dependencies limit effective immunotherapies
Macrophages regulate immune responses through mutually-exclusive activation states
Macrophages are co-opted by tissue damage signals
Macrophage TME accumulation predicts poor prognosis across huma
Two main sources for macrophages in tissues
Breast cancer macrophages originate from TRMs and mon
Macrophage education dynamics revealed by scRNA-seq
Immunophenotyping of SAMs vs TNMs in vivo
Spatial interactions determine phenotypic heterogeneity in human breast cancer macrophages
Summary
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